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- NSN 6550-01-606-4220
6550-01-606-4220
Description: He Ensyme Inhibitor
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Export Restrictions
Any part(s) quoted or sold by MilSpec.parts and re-sold (to a third party) CANNOT be sold to a USA embargoed country, company, entity, or person(s). Depending on the ECCN, these commodities may require export license or exception. Diversion contrary to U.S. law is prohibited. Unless otherwise noted, this sale, order or quote may include Munitions List Items (MLI) or Commerce Controlled List Items (CCLI). MLI/CCLI property is controlled by the US Government and in many cases cannot be transferred (exported, sold or given) to a foreign country, a non-US Citizen/National or a non- Permanent US Resident without a valid State/Commerce Department export authorization. It is the responsibility of (the Purchaser) to determine what the applicable requirements may be, to obtain all necessary authorizations, licenses or approvals and are subject to both review and acceptance by MilSpec.parts. The use, disposition, export and re-export of the property covered or included in this sale, order or quote is subject to the provisions of law referenced in End-Use Certificate DLA form 1822, DEC 2001, including, but not limited to, the Arms Export Control Act (22 USC 2751 et seq.); Export Administration Act of 1979 (50 USC App. 2401 et seq.) as contained under Executive Order 12924; International Traffic in Arms Regulations (22 CFR 120 et seq.); Export Administration Regulations (15 CFR 730 et seq.); Foreign Assets Control Regulations (31 CFR 500 et seq.) and the Espionage Act (18 USC 793 et seq.)
6550-01-606-4220 Properties
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NSN:6550-01-606-4220
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Description:He Ensyme Inhibitor
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Shelf Life:N/A
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DEMIL:NO
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UOM:--
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Part Name Assigned By Controlling Agency:Z (ii) potoporphyrin ix
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Special Features:A potent and selective inhibitor of heme oxygenase, the enzyme which generates carbon monoxide (co) and biliverdin. Inhibits soluble guanylyl cyslase and produces a time- and concentration-dependent inactivation of all three isoforms of nitric oxide synthase. Inhibits relaxation induced by acethycholine, anp, and vip in isolated rat aorta. Blocks interleukin-1 and prevents induction of long-term potentiation. Also useful as an indicator of iron deficient erythropoiesis. Does not cross the blood-brain barrier.
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